written by Sarah Howard
Coordinator of the Diabetes-Obesity Spectrum Working Group
Over 18,000 people from around the globe gathered in Boston June 5-9, 2015, for the American Diabetes Association’s premier annual scientific conference. Thanks to CHE, I was able to attend, and here summarize information I found on the development of diabetes—including environmental factors (especially chemicals), developmental origins, and the natural history of the disease.
While there were not any sessions on environmental chemicals per se, I did find ten posters on this topic (see below for links to abstracts and online e-posters). The one that struck me most was by Su Hyun Park, who found an association between levels of persistent organic pollutants (POPs) and beta cell dysfunction in 7-9 year old Korean children. Exposure to POPs, as well as to other chemicals, have been associated with beta cell dysfunction in other studies before, but there are few studies in humans, even fewer in children, and few at exposure levels found in the general population. (I told her I thought that hers was the most important poster of all 2373 of them and she laughed, but I stand by my opinion).